Tag Archives: rodenticides
Updated: 1 August 2014.
“More people have died because of chemicals than during
World War II” (Ollivry 2013).
2.0 ERRONEOUS INDUSTRIAL & INSTITUTIONAL IMPERATIVES
a/ long-term TOXIC outcomes and
3.1 WE (THE PEOPLE) DIDN’T THOROUGHLY KNOW
• lead in paint;
• Agent Orange;
• flouride (see: http://dianabuckland.webs.com/);
• mercury fillings
• NSAIDs or Advil/Aleve/Ibuprofen/Naproxen, Cox-2 Inhibitors or Celebrex/Celecoxib (Collins 2011);
• Vioxx, etc.
For example, the European Union recommends the use of a commonly-used safety factor of 100.
But, says Kefford, the latest evidence suggests that this safety factor is too small (Salleh 2012).
* decreased short term memory,
* poor concentration,
* altered emotional states (emotional lability, often oscillating between anxiety and depression).
3.2 WE DON’T WANT TO KNOW
3.2.1 BURIED PROBLEMS
Why did the discipline of toxicology have to change in recent years?
The next part of this same quote is wrong:
The new toxicology regime will involve:
Or, in more detail:
In the US:
In Tasmania (Australia):
Small wonder that the following is typical of our chemical experience:
Clearly, the armchair idealogues working with the FSC have bastardized the science (Tomkins 2013).
Or, put slightly differently, the Precautionary Principle:
So, how does Big Biotech manage to sideline the Principle?
“There’s no conclusive link” means… We don’t want to know, we don’t want YOU to know.
3.3 THE SLOW, CONSISTENT DEATH PLAN/STRATEGY IN IT’S MANY FORMS
3.3.1 META TREND: BACKGROUNDER
3.3.2 META TREND: TOXIC AG’S BAD SCIENCE & BAD BEHAVIOR
Another 200 chemicals are known to cause clinical neurotoxic effects in adults. [p.2167]
And the failures have not just occurred in India:
compromised by the introduction of GM crops. Now, the Government wants to find out what levels of contamination are acceptable.
And from the actual report, this:
And when farmers and farms fail, it is easier to come in and grab the land:
Corporate fangs dripping blood… And, even more to the GMO point:
And, even MORE to the utterly irresponsible and corrupt conflicts of interest GMO dynamic:
Continuing the above quote:
And, even MORE to THE health authority and corporate link theme:
And the wider environment consideration:
AND, EVEN MORE TO THE GMO POINT:
3.3.3 META TREND: INDUSTRIAL TOTALITARIANISM
And, in consideration of how fascist the new Biotech is becoming:
Shell and Chevron are in the spotlight this week, with shareholder meetings and a historic trial.
What does Monsanto DO? What are its characteristics, it’s ‘legacy’?
• Aspartame or NutraSweet, Equal (causes 94 health issues)
• Roundup (toxic) • Petroleum-Based Fertilizer (destroys soil integrity)
• Polystyrene (5th worst generator of hazardous waste)
• Saccharin (carcinogenic)
• PCBs (Cancer: Monsanto knew about PCB dangers from 1956 on…)
• DDT (toxic) • Dioxin (toxic)
3.3.4 META TREND REFORMATION?
3.3.5 CAN A BAD META TREND PRODUCE ANYTHING OTHER THAN BAD THINGS?
Despite the above, isn’t GMO technology just simply somehow “better” in some way?
Lower yields not a problem for you on your farm? Look at your children, and then read this:
3.3.6 THE DEATH PLAN SPREADS… DEATH!
3.3.7 A BRIEF CURE
What type of farming should we ultimately strive for?
3.4 A NEEDED SAVING FAST CHANGE EVENT (THE MEANING OF EVIL)
The math is straight forward. One in four will perish.
Could it get any worse? What about an ‘enhancement’ of the current global HIV pandemic storm?
for one with its destruction of the ecosystem, chemical intoxication of the population and
reliance on immune-suppressing medications and food additives.
creates a “perfect storm” for the spread of infectious disease. (Adams 2014)
4.0 GOOD CONNECTIONS USED BADLY BY PESTICIDE TECHNOLOGIES
Ultimately, pesticide/herbicide use:
6. Creates monstrous disease scenarios via increased use through herbicide tolerant GMOs:
5.1 NATURE INVASION, GENETIC ‘ENHANCEMENT’, PATHOGEN/TOXIN RELEASE & EXPOSURE, & ACCIDENTS
5.1.1 NATURE INVASION
And, please slowly absorb this quite spectacular assessment:
As a preface:
engineered eucalyptus tree.
gum. Each “transgenic” cell has already re-generated into a young river red gum, growing
in sterile conditions in a laboratory.
scientifically confirmed genetic transfer into the river red gum.
Forestry and Plant Industry is simply a “marker” to show that the technology works.
(Eucalyptus nitens and globulus) both of which are important commercial plantation trees
in Australia. (Keating 1992)
5.1.2 GMO ATTACK
On the third point above, please note:
If the above is bad, then this is much worse because numerous experts:
Now, let us contrast GMO transgene escapes to pesticide:
5.1.3 EXTENDED GMO SPECIFICS: ENDOTOXIN & OTHER ATTACKS
184.108.40.206 ENDOTOXIN, PARTICULATE/POLLEN, GASEOUS & FUNGAL BIOAEROSOLS
The analogue of the above occupational exposures in an open/external environment could be:
• Storms and high winds (including trees blown down or branches snapped off)
• Harvesting operations
• Post harvesting cleanup
In regard to the last point, please note:
…stomatal opening also influences the emissions of a range of terpenes…
And, by extension, the above particulates will contain GMO endotoxin that will escape AND pollute:
crops are currently practised to control infestations of crop-eating insects. The
increasing use of these biopesticides could lead to an increase in Cry1Ab endotoxin in
both terrestrial and aquatic environments (Douville, et. al. 2005).
endotoxin represent an increasing health problem (Togbe, et.al. 2007:768).
Note further GMO Accident #2:
1.Horizontal gene transfer at plant-surface sites
Comment by Ignacio Chapela, Berkeley/TromsÃ¸
And here is an EXTREMELY interesting comment:
How widespread are industry exposures?
Why should we really worry?
Further, in terms of ecological attacks, it is known that:
Thirdly, and a little more specifically:
Put another way:
And in terms of tree plantations:
All the above means nothing less than mass transgene ESCAPE:
And here’s an indication of toxicity to Multiple Chemical Sensitive individuals (like me):
I am one of the MCS “some people”.
220.127.116.11 POLLEN INCLUSIONS
18.104.22.168 PESTICIDE INCLUSIONS
And a little more specifically:
22.214.171.124 BIOSOLIDS, RUNOFF, DUST & HUMAN EXPOSURES
126.96.36.199 HORIZONTAL Bt GENE TRANSFER ON THE HEELS OF TRANSGENIC MANIPULATION
Bt and CpT I genes can result in persistence of Cry1Ac and CpT I proteins and
negatively affect soil microbial and biochemical properties (Chen, et. al. 2011:67).
5.1.4 EXTENDED GMO GENERALITIES: BROAD PANDEMIC POTENTIAL & MASTER CONSPIRACIES
188.8.131.52 PANDEMIC POTENTIAL
And, in terms of GE trees:
184.108.40.206 MY SYMPTOMS
including sensitivities to a broad range of environmental chemicals such as solvents,
paints, terpenes, glues, cleaning chemicals, chlorine and chlorinated products, plasticisers,
fragrances and more. Of particular concern at present is Mr Thompson’s adverse
reactions to the specific terpenes found in Eucalyptus nitens plantations and the surrounding
area. His health has deteriorated markedly with worsening of his respiratory
and joint/muscle inflammation and pain. (Donohoe, Medical Letter, May 12, 2014)
220.127.116.11 COMMERCIAL CONTRIVANCES & PREDICTABLE OUTCOMES
deaths] there had been a helicopter crash carrying pesticides and herbicides in the
upper catchment, apparently aerial spraying trees. And this was the first that
Id heard about plantations in that area. When we started to look at the size of the
plantations it became apparent that here was a very large source of potential toxic
chemical (Scammell 2010).
18.104.22.168 THE HUMAN HEALTH BIO-TERRORISM EXPERIMENT: GMO MASTER CONSPIRACIES
22.214.171.124 BACTERIAL-ENDOTOXIN TESTING
1. be inhaled by animals and humans,
2. be underestimated in enumeration,
None of this is good news for GMO .
126.96.36.199 BRIEF: PLANTATIONS & ASTHMA
New Zealand’s incidence of asthma is similar to Australia’s…
Figure 6.01 shows a map of high asthma areas…
The areas in the North Island correspond with the forestry areas especially (Gibbs 2006:267-268).
2. the additional monumentally ignorant rush for assumed genetic superiority…
5.2 ECOLOGICAL RAPE & POLITICS
The politics of the matter:
pesticides: the Duck, Inglis, Bird, Jordan, Montagu, Prosser, Rubicon, South Esk,
George, Little Swanport, Macquarie, Great Forrester, Brumby Creek, Derwent and Liffey …forestry plantations are now growing in 44 of the States 48 water catchments. …Water testing by our state government is done sporadically and pesticide detections
rarely result in investigations to find their source
6.0 PESTICIDES’ MISAPPROPRIATED DESTINATIONS: SPRAY DRIFT & MIGRATION
Also, from Australia:
And following up after the above comment, an email:
Spray drift damaging a neighbor’s crops?
Drift and migration?
The source of phenoxy herbicides at control sites is unclear… (ibid.:49).
6.2 CHEMICAL MIGRATION INTO WATER PIPES
6.3 CHEMICAL MIGRATION INTO & THROUGH BUILDINGS
7.0 HERBICIDE RESISTANT WEEDS
8.1.1 CHEMICAL TOXICITY & PANDEMICS
And, more generally:
• multiple individual exposures to different chemicals (additive effects),
• exposures occurring over extended time,
• Tiny exposures adversely affecting those with MCS.
• Genetic AND epigentic effects from exposure
On the last point, note:
Back to glyphosate generally:
It is the human slate-wiper, the invisible ultimate death, the filovirus named Ebola…
And, what happens when superbugs are already resident in the hospital that you need to visit?
8.1.2 CHEMICAL PERSISTENCE & MOBILITY
Note the literature on glyphosate persistence and mobility:
The PERSISTENCE of glyphosate (and its consequent buildup over repeated sprayings) is supported:
8.1.3 OTHER HERBICIDES: PESTICIDE TOXICITY, MOBILITY & CONTAMINATION
Note the following:
188.8.131.52 Haloxyfop-R methyl ester in Verdict 520 Herbicide
Basic Tox of Haloxyfop: Risk of Serious Damage To Eyes
or haloxyfop-P methyl ester or with neat substances. The toxicokinetic studies
indicate that absorbed methyl ester will rapidly be hydrolysed to the parent acid and
the S-form haloxyfop present in racemic haloxyfop will instantaneously undergo
stereochemical inversion to haloxyfop-P. Therefore, the various compounds used for
testing are assumed to elicit the same systemic effects following administration and
these effects can be attributed to haloxyfop-P. The absorption is rapid (> 80%) and
the excretion extensive. The acute oral toxicity is moderate i.e. LD50 is around 300
mg/kg bw and the dermal toxicity low LD50> 2000 mg/kg bw, proposed classification of
Xn, R22 “Harmful if swallowed”. No acute inhalation toxicity studies are available.
Neither racemic haloxyfop nor haloxyfop-P methyl ester was irritating to skin and
haloxyfop-P methyl ester was not a sensitizer. Haloxyfop-P methyl ester is not
irritating to the eye whereas racemic haloxyfop induced signs of irritation in the
conjunctival sacs and iris and caused corneal opacity covering up to 100% of the
cornea in all animals. Signs of irritation (corneal opacity) persisted for 21 days in
un-rinsed eyes racemic haloxyfop is therefore irritating to the eye and the
classification of Xi; R41 “Risk of serious damage to eyes” is proposed. The relevant
short term NOAEL is 0.5 mg/kg bw/day based on the 1-year dog study which would also be
said to cover the effects observed in the 90-day studies in the dog and monkey at 2
mg/kg bw/day (EFSA Journal 2009:3).
AOEL (Acceptable Operator Exposure Level)
respectively. The AOEL is exceeded (169%) according to the UK-POEM even with PPE
(coverall) but is below according to the German model if PPE (coverall and gloves) is
applied (12%). The estimated worker and bystander exposure is below the AOEL (ibid.).
Risk Assessment for Herbivorous and Insectivorous Birds and Mammals
values above the Annex VI trigger indicating a low risk. For medium herbivorous and
insectivorous mammals the acute risk is considered to be low, while a first tier high
long-term risk was identified. The Member State experts in EPCO 22 did not accept a
proposed refinement using a higher endpoint from a 16-week dietary study. It was
agreed to use the endpoint of 1 mg/kg bw/day from a 2-generation reproduction study.
Furthermore, since the half-life for residues in vegetation was observed to be longer
than the default value, residue decline data for each crop should be used in the risk
assessment. The resulting TER values were foreseen to be below the Annex VI trigger
indicating a high risk… (ibid:5).
They may also cause liver and kidney damage…
ethoxyethyl from days 6 to 16 of pregnancy reduced the number of live offspring per
litter and caused vaginal bleeding in the mother…
between days 6 and 16 of pregnancy caused developmental abnormalities in the
offspring’s urogenital system and death to the fetus…
pregnancy caused delayed bone formation in the offspring…
adult rats (8). Doses of 0.6 mg/kg/day for 2 years in mice caused reduced body weight
gains and increased liver weights in mice. In dogs, 5 mg/kg/day causes a significant
decrease in serum cholesterol, as well as a decrease in thyroid weight… (Extoxnet
The USEPA put it this way:
F1/F2b generation (USEPA 2012).
Aquatic Organisms Risk
sensitive group of organisms (EFSA Journal 2009:6).
0.54 mg/l for fathead minnows, 0.28 mg/l for bluegill sunfish, and 1.8 mg/l for
rainbow trout. The LC50 (48 hours) for Daphnia is 4.64 mg/l (6) (Extoxnet 1996).
184.108.40.206 Clopyralid 750g/kg (Group I) in Clomac Forestry Herbicide and Other Products
Environmental Efects: Toxic to some beneﬁcial insects. It is very persistent and
moves quickly through soil…
…is slightly toxic to birds…
Changes to liver and kidney weights as well as, hyperplasia of the stomach lining were
found in rats fed moderate to high doses in a 2-year study… A one year study with
dogs showed increased liver weights and decreased red blood cell count at all but the
lowest dose tested… Another study found that weights of rabbit fetuses decreased at
both low and high doses of clopyralid. Skeletal abnormalities were also observed in
these fetuses at all doses and at the highest dose, accumulation of excess fluid
around the brain was evident, which resulted in small brain and enlarged skulls…
species that are vulnerable to clopyralid, including five species of rare cactus…
not readily bind to soil and is highly soluble in water. As a result, it is very
mobile in soil and has the potential to move towards groundwater and contaminate
surface water. Clopyralid has even been found in soil water samples taken at depths of
6 feet, up to 30 days after initial surface treatment… Since microbial activity
decreases with depth, clopyralid has the potential to persist longer at lower soil
depths. Persistence of clopyralid in vegetation was evidenced by the contamination of
composts from harvested lawn and grass clipping sprayed with clopyralid. Clopyralid
has been detected in vegetation 365 days after initial treatments…
compost and manure in Spokane, Washington. Upon investigation it was found that high
concentrations of clopyralid were present in composts made from lawn clippings,
straw, and leaves collected by curbside programs… The Spokane area had a large
residential population that utilized lawn care services, most of whom applied
clopyralid as part of their weed control scheme. Clopyralid, which does not breakdown
during the composting process, stunts the growth of crops such as potatoes, tomatoes,
peas, beans, leuce, pepper, alfalfa and sunflowers at very low levels- even as low
as 1ppb… Organic farmers were also severely affected since they rely of manure and
composts to supply soil fertility. In 2002, the state of Washington banned the use of
clopyralid on lawns and turf in order to keep the chemical from contaminating compost
supplies… That same year, California found that 65% of the composts samples tested
positive for clopyralid, which led to the cancellation of residential uses for
clopyralid in the state (Beyond Pesticides / NCAMP, no date).
inhalation of ambient air, and ingestion of drinking water (Toxnet 2013).
8.2.2 STEAM & FLAME
8.2.3 CORN GLUTEN MEAL (CGM)
For excellent research on CGM, see: http://www.hort.iastate.edu/research/gluten.
8.2.4 KITCHEN RECIPES
8.2.5 ORGANIC HERBICIDES 1: OIL-BASED HERBICIDES
8.2.6 ORGANIC HERBICIDES 2: INCLUDING CLOVE, CINNAMON & COTTONSEED OIL
8.2.8 STEAM ON STEROIDS
9.0 TRUE SCIENCE
We cannot coexist with PESTICIDES and GMOs!
We’re fighting for our right to live! (Emmerich & Devlin 1996)
http://cabecahealth.com/wp-content/uploads/2013/05/FoodPlaguePrimer.pdf; accessed: 25
References cited in the quotes:
1. From a brochure Warning! Poisoned water? Authorised by Dr. Andrew Lohrey and widely distributed in Tasmania;
2. Alison Bleaney, ‘Chemicals: The dismal failure’ Tasmanian Times, 7 May 2012;
3. Andrew Lohrey, ‘Panel report dubious: designed to stifle public debate,’ Tasmanian times,2 July 2010, http://tasmaniantimes.com/index.php?/article/george-river-toxins-rerport-dubious-designed-to-stifle-debate/;
9. Tim Flannery, Here on Earth, Text Publishing, Melbourne, 2011, p. 168.
10. Alison Bleaney, ‘Chemicals: The dismal failure’ Tasmanian Times, 7 May 2012. http://tasmaniantimes.com/index.php?/article/how-chemicals-affect-us/.
OP09: Pest, Disease, and Weed Management; Source: http://ricecongress.com/previous/extPdfs/OP09-3867-Cuong-fgh-edited.pdf; accessed: 1 Mar. 2014.
References cited in the quote:
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Saxena, D., Flores, S. & Stotzky (1999) Insecticidal toxin in root exudates from Bt corn. Nature 402: 480.
Speight, M.R., Hunter, M.D. & Watt, A.D. (1999) Ecology of insects: concepts and applications. Blackwell Science, Oxford.
Tabashnik, B.E., Cushing, N., Finson, N., & Johnson, M.W. (1990). Field development of resistance to Bacillus thuringiensis in diamondback moth (Lepidoptera: Plutellidae). Journal of Economic Entomology 83:1671-1676.
Tabashnik, B.E. (1994). Evolution of resistance to Bacillus thuringiensis. Annual Review of Entomology 39: 47-79.
Talekar, N.S., & Shelton, A.M. (1993) Biology, ecology and management of the diamondback moth. Annual Review of Entomology 38: 275-315.
Tang, J.D., Gilboa, S., Roush, R.T. & Shelton, A.M. (1997) Inheritance, stability, and fitness of resistance to Bacillus thuringiensis in a field colony of Plutella xylostella (L.) (Lepidoptera: Plutellidae) from Florida. Journal of Economic Entomology 90: 732-741.
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 Laitinen, P. et al. (2009) Glyphosate and phosphorus leaching and
residues in boreal sandy soil. Plant Soil 323, 267–283.
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glyphosate in selected Canadian boreal forest soils. J. Agric. Food
Chem. 37, 437–440.
 Laitinen, P. et al. (2007) Glyphosate translocation from plants to soil –
does this constitute a signi?cant proportion of residues in soil? Plant
Soil 300, 51–60.]
Unknown Etiology in Sri Lanka?”; in Int. J. Environ. Res. Public Health 2014, 11, 2125-2147; doi:10.3390/ijerph110202125; Source: Personal Email from Channa Jayasumana, 27 Feb. 2014.
http://www.sourcewatch.org/index.php/File%3AKeating_Statement_on_GE_Eucalyptus_trees_5_August_1992.pdf; Accessed: 24 June 2014.
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7 Laumbach, R.J. and Kipen, H.M. (2005). Bioaerosols and sick building syndrome: Particles, inflammation, and allergy. Current Opinion in Allerg y and Clinical Immunolog y, 5(2), 135-139.
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 National Research Council (2000) Genetically Modified Pest-Protected Plants: Science and Regulation (Natl Acad Press, Washington, DC).
 Sears, M.K., R.L. Hellmich, D.E. Stanley-Horn, K.S. Oberhauser, J.M. Pleasants, H.R. Mattila HR, B.D. Siegfried and G.P. Dively Impact of Bt corn pollen on monarch butterfly populations: A risk assessment Proceedings of the National Academies of Science USA, 2001 Volume 98, pages 11937–11942.
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1. The amazing power of Chemical Diffusion:
2. Note the article: “Monsanto’s Bt Cotton Kills the Soil as Well as Farmers”:
Bacteria were reduced by 14%. The total microbial biomass was reduced by 8.9%.
3. Note, in terms of a urine and water test for glyphosate:
4. How uncontaminated is your water?
MY CHILD & I ARE SICK
AND YET TOXIC CHEMICALS ARE STILL FREELY SPRAYED
WORLDWIDE DISEASE PANDEMICS FROM PESTICIDE
PO Box 605
New Norfolk TAS 7140
8 April 2012
Web sites: http://poisonedpeople.com
And, on a Chernobyl and Iraq nuclear note:
§10. Whereas children are the most vulnerable and exposed to contamination by these pollutants…
PESTICIDE DELETES POTENTIAL ON ALL FRONTS
THE TARGET ORGAN FOR PESTICIDES IS
(Dr. Mark Donohoe, 2001, pers. comm.).
I AM JUST TRYING TO PROTECT MY CHILD. WHAT COULD POSSIBLY BE WRONG WITH THAT!?
WOULDN’T YOU WANT TO MAKE THESE SPECIAL LITTLE PEOPLE SAFE?
WHAT IS ILLEGAL IN WAR MUST BECOME ILLEGAL IN PEACETIME!
 (Royal Society of Chemistry 2009).
ROUNDUP, THE MAJOR CAUSE OF, NOW, THE WORLD’S MOST POPULAR CANCER!!?
WHAT’S CHILDHOOD ASTHMA WORTH?
WHAT’S ADHD WORTH?
WHAT’S CANCER WORTH?
WHAT ARE MISCARRIAGES WORTH?
What rate increases IN DISEASES (or behavioural problems) might we find in McQuade Avenue?
QUITE THE CONTRARY [xliii]
Back to The Paris Appeal:
Measure # 1
Measure # 2
Measure # 3
Measure # 4
Politicians of Australia & world leaders alike, WE NEED THOSE LAWS. FIFTY YEARS AGO.
Feel free to write or email me should you wish to follow up on this issue.
NEW SOUTH WALES CO-COORDINATOR, MCS-GLOBAL WEBSITE: www.mcs-global.org
THIS LETTER HAS BEEN SENT TO:
Ford, J., 2008, Dr. Judy Ford Interview, Channel 10 News, Monday 29th December 2008.
Grabau, B.J. (Dr. Brendan), 2005, Toxicology Report: Mr. Murray Thompson.
Kerr, P., 1995, ‘Toxic tip to close’, Penrith Press, April 25, 407 High Street PENRITH NSW 2750.
Liedloff, J. 1986, The Continuum Concept, Penguin Arkana, Harmondsworth, Middlesex, England.
AN ENTIRE PLANET IN EXTREMIS.
 All employees have a right to “work in an environment that is safe” (ACTU, 2008).
 Synthetic pyrethroids are used for termite control in public housing.
 Sinclair & Pressinger, no date, citing Bell, et al., 2001; my emphasis.
 DirectGov, 2007.
 American Cancer Society, 2008.
 National Center for Health Statistics, 2008.
 CureResearch.com, 2007.
 NKDEP, 2005.
 Mindfully.org, 2007.
 Bio-Medicine.org, 2008.
 Yb Dato Liow Tiong Lai, 2008.
 Clennell, 2004.
 “Another 200 chemicals are known to cause clinical neurotoxic effects in adults. [p.2167]
 A “continuum of expectations” (Liedloff 1986).
By Murray Thompson (BAppSci Environmental Health 1998, University of Western Sydney)
1.0 HUMAN HEALTH IMPACTS
Pesticides promote the classic symptom of abdominal pain in children (Dr. B. Grabau, 2005, pers. comm.), or, “non-specific abdominal gastric pain” (Dr. B. Grabau, 2005, Private Medical Report). This means that many aches and pains children get may not simply just be “normal”.
“Hundreds of thousands of people are dying around the world each year from the effects of the use, or misuse, of pesticides” (Konradsen, et al., 2003).
“Doctors at a weekend conference in Winnipeg say there is a disturbing trend when it comes to the rising rate of certain cancers. They say pesticides are to blame for the increase – especially in childhood cancers” (Sinclair & Pressinger, no date, citing Winnipeg CBC News – June 7, 2004).
A “study showed there was approximately [a] 2-fold greater risk of having a
stillbirth if the mother lived within 1 mile from an agricultural area which used
organophosphate – pyrethroid – carbamate – or chlorinated pesticides” (Sinclair & Pressinger, no date, citing Bell, et al., 2001).
workers recovering from organophosphate poisoning… Among workers who apply organophosphates but have not suffered poisoning episodes, some studies have shown similar types of subclinical neurobehavioural deficits and subclinical electroencephalographic abnormalities…” (O’Malley 1997).
2.0 ENVIRONMENTAL & ANIMAL-DISEASE IMPACTS
“Glyphosate spray drift from both ground and aerial applications has been measured from 400 to 800 meters from the target site… Drift that is one thousand times less than the usual application rates has been shown to damage surrounding vegetation, including the killing of wild plants. This is an important reason why it should not be used in national parks and environmentally sensitive areas for weed control” (Leu 2007).
Glyphosate [Roundup] is persistent (NCAP, 2000:2, citing US EPA, 1993-2) and may last up to 3 years (NCAP, 2000:2, citing Torstensson et al., 1989), while its metabolite, AMPA [aminomethylphosphonic acid], may persist even longer, “with a half life in soil between 119 and 958 days” (Buffin & Jewell, 2001, citing WHO, 1994).
Roundup has been heavily implicated in thyroid, liver and pancreatic tumours in test animals (Cox, 1993:4, citing Dykstra & Ghali, 1991). Roundup is also now shockingly linked to Mad Cow’s Disease (Rotella 2003), and to an increase in plant diseases (gmwatch.org 2010).
http://www.beyondpesticides.org/schools/sepa/SEPA_fact&figures.htm; accessed: 18 July 2008.
http://www.cell.com/AJHG/abstract/S0002-9297(08)00143-2; accessed: 26 June 2009.
http://www.sciencealert.com.au/news/20082608?17859?2.html; accessed: 20 Sept. 2008.
http://oncolink.org/types/article.cfm?C=10&s=36&ss=820&id =9539; accessed: 16 June 2008.
By Murray Thompson (BAppSci Environmental Health 1998, University of Western Sydney)
Essay URL: https://poisonedpeople1.wordpress.com/2012/11/07/
Short link: http://wp.me/p2DVqC-3t
And the Piper Alpha North Sea oil platform disaster.
And the “Triangle Shirtwaist Factory fire in New York City” in 1911 (more than 100 died).
And the “Kader Toy Factory fire” in Thailand in 1993 (188 workers died, mostly young women).
And the 1906 “Courrières mine disaster in Courrières, France” which killed 1,099 workers.
And the “Benxihu Colliery disaster in Benxi, Liaoning, China” which killed 1,549 workers.
And the Port Chicago munitions explosion of 1944.
And the 1962 Centralia, Pennsylvania coal mine fire (still burning today!).
And… my son’s and my (http://indiegogo.com/poisoned-people) pesticide poisoning.
25th April 2010.
TITLE (Note: This is an online Campaign)
The Human Right To NO PESTICIDE POISONING!
Short link: http://wp.me/p2DVqC-3o
Note: “Most tragically, suffering, illness and disease surround us today in a way we would not have imagined a half century ago. We have banished some diseases only to have them replaced by a grumbling yet profound toxicity which is stripping our children of their rightful future” (Donohoe, 1998:38).
My Son & I Poisoned
My son (at age 8) and I were both poisoned (me severely) when we had two pesticide treatments forced on us by the New South Wales Department of Housing (see: http://poisoningandlegalaction.com.au). I, the pesticide injury victim, was blamed wholly by Housing’s Barrister in my trial for our poisoning! See: http://www.indiegogo.com/poisoned-people.
What Do We Need To Do?
We, the people, need to demonstrate true character and tell our politicians that we fiercely object to the present corruption of our legal and political systems in their protection of authorities and government departments from claims of negligence. We want fairness in the assessment of such claims, and we want our basic Human Rights back.
http://caselaw.lp.findlaw.com/data/Constitution/article04/, or http://www.usconstitution.net/const.html#A4Sec4.
The Paris Appeal
“We call upon national decision-makers, European Authorities, international organizations, and specifically the United Nations Organization (UNO), to take the following measures:
Banning all products that are certainly or probably carcinogenic, mutagenic or reprotoxic (CMRs) for human beings, as specified by competent international scientific authorities and organizations, and therefore applying to these products the principle of substitution; exceptionally, whenever implementation of this principle is not feasible and the use of the product concerned is considered unavoidable, limiting its use to a minimum with particularly stringent measures of fixed quotas,
Applying the precautionary principle to all chemicals that, regardless of toxicity characteristics specified in Measure # 1 (refer to §9 and §13), and because they are persistent, bioaccumulative, toxic (PBT) or very persistent and very bioaccumulative (vPvB), constitute an allegedly serious and/or irreversible danger for human and/or animal health, and more generally the environment, without waiting for the definite proof of an epidemiological link, so as to anticipate and avoid serious and/or irreversible sanitary or ecological damage,
Promoting the adoption of toxicological standards or international thresholds to protect people, based on the assessment of risks for the most vulnerable, i.e. mostly children and the embryo.
With respect to the precautionary principle, adopting programs with scheduled deadlines and targets in precise figures so as to achieve elimination or strictly regulated reduction in polluting substances emissions and in the utilization of marketed chemicals, such as pesticides, modeling the reduction in use implemented in Sweden, Denmark or Norway…” (IDEA, 2006).
What Do We Really Want? Health & Perpetuity!
We want our children to be safe from the transnational chemical giants-sponsored, anti-Duty of Care, anti-Precautionary Principle, unexamined and utterly irresponsible, CRIMINAL and worldwide love-affair with toxic and reproductive capacity (fertility)-destroying chemicals. We want this Extinction Level Event stopped! Think this statement is too crazy? Note:
the effects of the use, or misuse, of pesticides” (Konradsen, et al., 2003).
stillbirth if the mother lived within 1 mile from an agricultural area which used organophosphate – pyrethroid – carbamate – or chlorinated pesticides” (Sinclair & Pressinger, no date, citing Bell, et al., 2001).
§10. Whereas children are the most vulnerable and exposed to contamination by these pollutants…
§11. Whereas these polluting substances or products can induce diseases in children population, such as those listed in §9; whereas one out of seven children in Europe suffers from asthma, whereas asthma is made worse by city and home pollution; whereas incidence in pediatric cancers has been on the rise for the last 20 years in some industrialized countries; and whereas these considerations all lead to the fact that Children are now in serious danger…” (IDEA 2006).
(NCAP, 1999:3; Bonn, 2005; Cox, 2004).
http://web.mac.com/doctormark/DoctorMark/KUS.html; accessed: 13/12/2008.
http://www.ideaireland.org; accessed: 10th October 2007.
THE ‘LEGAL’ AND ‘SCIENTIFIC’ DESTRUCTION OF HUMAN RIGHTS, PUBLIC HEALTH & PLANET EARTH
greatly to the downfall of states, and is a sign of rot within a political system” (Aulich 2006).
By Murray Thompson (BAppSci Environmental Health 1998; Hons I Social Ecology 1999, University of Western Sydney)